Rather
than people lying in their beds in romantically pale and waiting to
die, the reality was that there were thousands of infected people.
But tuberculosis was a selective killer. A study of urban postmortem
examinations in the 1890s showed that the majority of adults were
infected by tuberculosis but only ten percent went on to develop
symptoms and die of the disease. Tuberculosis could lay dormant and never become active or it could
activate and kill within weeks or disappear and come back again. In
England active cases of tuberculosis had been at their peak from 1790
to 1840, in lock step with the Industrial Revolution which ensconced
people in poorly ventilated factories for long hours and housed them
in inadequate tenements, both breeding grounds for disease.
Although
tuberculosis incidence was on the downswing by 1840, it was still a
major killer. While poor and densely populated areas had higher rates
of the disease so did certain occupations such as bakers, tailors,
printers, seamstresses and shoemakers; where industrial processes
produced dust or vapours which would affect the lungs.This
occupational hazard fit in with the first tuberculosis related death
my grandfather's family. In 1888 his uncle, John Thomas Chambers,
died from phthisis. He was a shoe manufacturer and was 34 when he
died. Had his occupation triggered a genetic predisposition to the
active form of the disease and had he brought it home to his family?
Sources
Thomas
Dormandy, White Death: A History of Tuberculosis. (New York: New York University Press, 2000).
F.B.
Smith, The Retreat of Tuberculosis 1850-1950. (London, New York, Sydney: Croom Helm, 1988).
Michael Worboys, “Before McKeown: Explaining the Decline of Tuberculosis in
Britain, 1880-1930.” In Tuberculosis Then and Now, ed Flurin Condrau and
Michael Worboys (Montreal & Kingston: McGill-Queen's University Press,
2010).
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